In obstructive jaundice, no bilirubin reaches the small intestine, meaning that there is no formation of stercobilinogen.
2.
In this case, presence of bilirubin ( conjugated ) in the urine without urine-urobilinogen suggests obstructive jaundice, either intra-hepatic or post-hepatic.
3.
Complications from delayed surgery include pancreatitis, empyema ( " collection of pus " ) or perforation of the gallbladder, cholecystitis, cholangitis, and obstructive jaundice.
4.
Low urine urobilinogen may result from complete obstructive jaundice or treatment with broad-spectrum antibiotics, which destroy the intestinal bacterial flora . ( Obstruction of bilirubin passage into the gut or failure of urobilinogen production in the gut .)
5.
Obstructive jaundice reduces biliary bilirubin excretion, which is then excreted directly from the blood stream into the urine, giving a dark-colored urine but with a paradoxically low urobilin concentration, no urobilinogen, and usually with correspondingly pale faeces.
6.
In a patient with a history of lung cancer presenting with jaundice and a non-palpable gallbladder it is likely that it is caused by a double cancer affected the common hepatic ducts and not from metastasis to the lymph node causing obstructive jaundice.
7.
These parasites are long-lived and cause heavy chronic infections that may lead to accumulation of fluid in the legs ( edema ) and in the peritoneal cavity ( ascites ), enlarged non-functional gall-bladder and also cholangitis, which can lead to periductal fibrosis, cholecystitis and cholelithiasis, obstructive jaundice, hepatomegaly and / or fibrosis of the periportal system.
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