In fresh ( rather than " in vitro " fertilization, final oocyte maturation triggering with GnRH agonist instead of hCG decreases the risk of ovarian hyperstimulation syndrome but decreases live birth rate.
12.
"Coasting ", which is ovarian hyperstimulation without induction of final maturation, does not significantly decrease the risk of ovarian hyperstimulation syndrome ( OHSS ), according to a Cochrane review of randomized trials.
13.
Less common causes include esophageal rupture or pancreatic disease, intra-abdominal abscesses, rheumatoid arthritis, asbestos pleural effusion, mesothelioma, Meigs'syndrome ( ascites and pleural effusion due to a benign ovarian tumor ), and ovarian hyperstimulation syndrome.
14.
A small number of underpowered randomized trials came to the result that use of clomifene in addition to gonadotropins results in similar live birth rate but with a reduction in the incidence of ovarian hyperstimulation syndrome.
15.
Using a GnRH agonist instead of hCG for inducing final oocyte maturation and / or release results in an elimination of the risk of ovarian hyperstimulation syndrome, but a slight decrease of the delivery rate of approximately 6 %.
16.
Additionally, a systematic review and meta-analysis concluded that prophylactic treatment with cabergoline reduces the incidence, but not the severity, of ovarian hyperstimulation syndrome ( OHSS ), without compromising pregnancy outcomes, in females undergoing stimulated cycles of in vitro fertilization ( IVF ).
17.
Final maturation induction using a GnRH agonist is recommended in women with cancer undergoing fertility preservation, because ovarian hyperstimulation syndrome is associated with an increased risk of arterial thrombotic events such as stroke, myocardial infarction and peripheral arterial embolism, and this risk can add to an already increased risk caused by the cancer.
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