When there is insufficient nutrient minerals or osteoblast dysfunction, the osteoid does not mineralize properly, and it accumulates.
12.
This malignancy most often develops in adolescence during periods of rapid osteoid formation ( commonly referred to as growth spurts ).
13.
Another very rare condition is a disturbance in primitive transformed cells of mesenchymal origin which exhibit osteoblastic differentiation and produce malignant osteoid.
14.
Regions of developing, unmineralized bone ( osteoid ) may expand and encroach on the marrow space, resulting in myelophthisic anemia.
15.
Osteoblasts can also be stimulated to increase bone mass through increased secretion of osteoid and by inhibiting the ability of osteoclasts to break down osseous tissue.
16.
These include revascularization and formation of granulation ( scar ) tissue, absorption of necrotic fragments, intertrabecular osteoid deposition, and remodeling of new bone.
17.
Increased secretion of osteoid is stimulated by the secretion of growth hormone by the pituitary, thyroid hormone and the sex hormones ( estrogens and androgens ).
18.
The main reason for radiographic examination is to exclude bony lesions such as occult fractures, slipped upper femoral epiphysis or bone tumours ( such as osteoid osteoma ).
19.
Examples of benign bone tumors include osteoma, osteoid osteoma, osteochondroma, osteoblastoma, enchondroma, giant cell tumor of bone, aneurysmal bone cyst, and fibrous dysplasia of bone.
20.
During bone formation, an osteoblast is left behind and buried in the bone matrix as an " osteoid osteocyte ", which maintains contact with other osteoblasts through extended cellular processes.
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