Most acyl-CoA dehydrogenases are ? 4 homotetramers, and in two cases ( for very long chain fatty acid substrates ) they are ? 2 homodimers.
22.
This gene encodes the alpha subunit of the mitochondrial trifunctional protein, which catalyzes the last three steps of mitochondrial beta-oxidation of long chain fatty acids.
23.
In more detail, it is a disorder of peroxisomal fatty acid beta oxidation which results in the accumulation of very long chain fatty acids in tissues throughout the body.
24.
CYP2U1 metabolized arachidonic acid, docosahexaenoic acid ( DHA ), and other long chain fatty acids which suggests that CYP2U1 may play a role in brain and immune functions.
25.
While there is an accumulation of very long chain fatty acids in the brain, it does not seem to be the reason behind the disease as gene therapy does not correct it.
26.
Long chain fatty acids are negative allosteric regulators of ACC and so when the cell has sufficient long chain fatty acids, they will eventually inhibit ACC activity and stop fatty acid synthesis.
27.
Long chain fatty acids are negative allosteric regulators of ACC and so when the cell has sufficient long chain fatty acids, they will eventually inhibit ACC activity and stop fatty acid synthesis.
28.
Substrate length for mDECR catalysis is thought to be limited at 20 carbons, at which the [ very long chain fatty acid ] is first partially oxidized by pDECR in the peroxisome.
29.
CD36 recognises oxidized low density lipoprotein, long chain fatty acids, anionic phospholipids, collagen types I, IV and V, thrombospondin and " Plasmodium falciparum " infected erythrocytes.
30.
It causes an important accumulation of very long chain fatty acids, that can be treated with oils enriched with 24 : 1 ( Lorenzo's oil, Lunaria's oil ).
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