With a sufficient number of channels opening at once, the inward flow of positive charges carried by Na + ions depolarizes the postsynaptic membrane sufficiently to initiate an action potential.
22.
EPSPs and IPSPs compete with each other at numerous synapses of a neuron; this determines whether or not the action potential at the presynaptic terminal will regenerate at the postsynaptic membrane.
23.
And the structure of the synapse itself can be altered in any number of ways that weaken communication ( e . g . decreased number of neurotransmitter receptors on the postsynaptic membrane ).
24.
The activity-dependent synaptic responses also lead to rapid insertion of AMPA receptors into the postsynaptic membrane, which will act to maintain ongoing glutamatergic transmission as sustained calcium influx could result in excitotoxicity
25.
Cst1 ( CLSTN1 ) was first shown to be predominantly expressed in postsynaptic membranes of excitatory neurons, and were shown to have multi-purpose functions both within and outside the nervous system.
26.
A higher concentration of Ca 2 + enables synaptic vesicles to fuse to the presynaptic membrane and release their contents ( neurotransmitters ) into the synaptic cleft to ultimately contact receptors in the postsynaptic membrane.
27.
In addition to the selectivity of splice variants, the levels of neuroligins, neurexins, and other interacting proteins present on the pre-and postsynaptic membranes influence the differentiation and balance of synapses.
28.
"' End plate potentials "'( "'EPPs "') are the depolarizations of skeletal muscle fibers caused by neurotransmitters binding to the postsynaptic membrane in the neuromuscular junction.
29.
In the neuromuscular junction, the diseases will either act on the presynaptic membrane of the motor neuron, the synapse separating the motor neuron from the muscle fiber, or the postsynaptic membrane ( the muscle fiber ).
30.
?-Neurotoxins antagonistically bind irreversibly to nAChRs of skeletal muscles, thereby blocking the action of ACh at the postsynaptic membrane, inhibiting ion flow and leading to paralysis . nAChRs contain two binding sites for snake venom neurotoxins.
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