21. Prominent suprasternal and carotid pulsations may be noted secondary to increased left ventricular stroke volume . 22. It is also possible to quantify the stroke volume in real-time on a beat-for-beat basis. 23. Therefore the stroke volume does not drop enough that it completely negates the increased heart rate. 24. In other words, if the end-diastolic volume increases, there is a corresponding increase in stroke volume . 25. This quantity is referred to as stroke volume . 26. This increased demand is met by an increase in the stroke volume of the octopus heart. 27. The decreased stroke volume causes a decreased forward cardiac output and an increase in the end-systolic volume. 28. Because stroke volume decreases in certain conditions and disease states, stroke volume itself correlates with cardiac function. 29. A reduced stroke volume may occur as a result of a failure of systole, diastole or both. 30. Because stroke volume decreases in certain conditions and disease states, stroke volume itself correlates with cardiac function.
