31. The presence of gastrin stimulates parietal cells of the stomach to secrete hydrochloric acid ( HCl ) / gastric acid. 32. Additionally, elevated gastrin levels may be present in chronic gastritis resulting from " H pylori " infection. 33. Gastrin is transferred from a specific type of G cell in the gastric epithelium to the ECL cells by blood.34. Vagal stimulation of the peptidergic neurons, occurring simultaneously, leads to the release of gastrin -releasing-peptide. 35. "' Little gastrin I "'is a form of gastrin commonly called as gastrin-17. 36. "' Little gastrin I "'is a form of gastrin commonly called as gastrin-17. 37. "' Little gastrin I "'is a form of gastrin commonly called as gastrin -17. 38. Eventually, all the parietal cells are lost and achlorhydria results leading to a loss of negative feedback on gastrin secretion. 39. Other possible mechanisms include increased vagal activity and secretion of insulin and gastrin as well as improved parent-infant relationships. 40. D cells in the stomach contain CCKBR ( which respond to gastrin ) and M3 receptors ( which respond to Ach ).
