In rare cases, excessive prolongation of ventricular repolarization by azimilide can result in predisposition towards severe ventricular arrhythmias.
32.
Together with the rapidly activating K + current ( IKr ), IKs is important for human ventricular repolarization.
33.
On the other hand, potassium channel opening accelerates repolarization of the action potential, possibly inducing arrhythmic reentry.
34.
Mutations resulting in loss of function of K + channels can result in delayed repolarization of the cardiac muscle cells.
35.
Benign Early Repolarization that occurs in the inferior and lateral portion of the heart muscle is associated with ventricular fibrillation.
36.
The delayed rectifier potassium ion current is largely responsible for the repolarization of ventricular cardiac myocytes by permitting potassium efflux.
37.
Calcium is removed from the muscle cell cytoplasm independent of repolarization ( depolarization signaling and muscle contraction are independent processes ).
38.
This shortens the repolarization period, thus speeding the rate of depolarization and contraction, which results in an increased heartrate.
39.
Azimilide dihydrochloride is a chlorophenylfuranyl compound, which slows repolarization of the heart and prolongs the QT interval of the electrocardiogram.
40.
Potassium channels are responsible for repolarization of the cardiomyocytes therefore increasing the rate at which cells can depolarize and generate contraction.
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