AAV-mediated gene transfer of the full-length " Mybpc3 " ( defined as gene replacement ) dose-dependently prevents the development of cardiac hypertrophy and dysfunction in homozygous " Mybpc3 "-targeted knock-in mice.
42.
Healthy cardiac hypertrophy ( physiologic hypertrophy or " athlete's heart " ) is the normal response to healthy exercise or pregnancy, which results in an increase in the heart's muscle mass and pumping ability.
43.
In animal models of disease, S100A1 protein levels has been shown to be altered in beta adrenergic signaling, prevention from cardiac hypertrophy and heart failure, as well as prolonged survival relative to non-transgenic controls.
44.
The homozygous " Mybpc3 "-targeted knock-in mice, which genetically mimic the situation of severe neonatal cardiomyopathy are born without phenotype and soon after birth develop systolic dysfunction followed by ( compensatory ) cardiac hypertrophy.
45.
An epidermal growth factor produced by monocytes and macrophages, due to an affinity for heparin is termed HB-EGF . It has been shown to play a role in wound healing, cardiac hypertrophy, and heart development and function.
46.
It has also been suggested that the root cause of many heart ailments is cardiac hypertrophy, which in turn is caused by hypoxia due to atmospheric CO, particulate matter, and peroxyl acyl nitrates, which reduces ATP synthesis in cardiac mitochondria.
47.
However, in vivo evidence suggest that chronic activation of p38 MAPK activity triggers restrictive cardiomyopathy with limited hypertrophy, while genetic inactivation p38? MAPK in mouse heart results in an elevated cardiac hypertrophy in response to pressure overload or swimming exercise.
48.
TSPO as a biomarker is a newly discovered non-invasive procedure, and has also been linked as a biomarker for other cardiovascular related diseases including : myocardial infarction ( due to ischemic reperfusion ), cardiac hypertrophy, atherosclerosis, arrhythmias, and large vessel vasculitis.
49.
On November 1, the New York City Medical Examiners Office announced that Caminiti died from " acute intoxication due to the combined effects of cocaine and opiates ", but coronary artery disease and cardiac hypertrophy ( an enlarged heart ) were also contributing factors.
50.
Al-Shaaban's aspirations won him two regional awards for best scientific research in 2006 and in 2009 . His research about AKAPs association with cardiac hypertrophy lead to the publication of 2 articles in the Journal of Molecular and Cellular Cardiology in 2007 and 2009.
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