Studies measuring cerebral metabolic rate for glucose ( CMRglc ) and cerebral blood flow ( CBF ) have indicated an indirect measurement of synaptic activity.
42.
On the other hand, sudden or rapid rises in blood pressure may cause hyperperfusion and increased cerebral blood flow, causing increased intracranial pressure and cerebral edema.
43.
Studies with PET scanning and transcranial Doppler imaging have shown that changes in cerebral blood flow, such as hypoperfusion and vasospasm, can follow penetrating head injury.
44.
Since higher processes in the brain occur almost constantly, cerebral blood flow is essential for the maintenance of neurons, astrocytes, and other cells of the brain.
45.
The pH-stat method may result in loss of autoregulation in the brain ( coupling of the cerebral blood flow with the metabolic rate in the brain ).
46.
This regulation of cerebral blood flow is achieved primarily by small arteries, arterioles, which either dilate or contract under the influence of multiple complex physiological control systems.
47.
Normally, cerebral blood flow is maintained by an autoregulation mechanism that dilate arterioles in response to blood pressure decreases and constricts arterioles in response to blood pressure increases.
48.
Alternative techniques employ arterial spin labeling ( ASL ) or weighting the MRI signal by cerebral blood flow ( CBF ) and cerebral blood volume ( CBV ).
49.
Cerebral blood flow remains unchanged over a wide range of arterial pressures ( mean arterial pressure of 50 150 mmHg ) through a process termed autoregulation of blood flow.
50.
PET scan findings indicate cerebral blood flow decreases in the left medial part of the left temporal lobe and the limbic and frontal systems as suffering from developmental abnormality.
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