It is thought that this region is responsible for the pH-dependent conformational shift that causes bound LDL to be released in the endosome.
42.
Entry via the endosome guarantees low pH and exposure to proteases which are needed to open the viral capsid and release the genetic material inside.
43.
In a special endosome called the MHC II compartment, cathepsin S cleaves the invariant chain, leaving CLIP bound to the MHC II complex.
44.
Once the virus has successfully gained entry into the host cell, the endosome acidifies, which alters virus topology by causing capsid components to disband.
45.
Unique endosome targeting sequences have been found in the N terminus of annexins I and II, which would be useful in sorting of endocytotic vesicles.
46.
Retromer plays a central role in the retrieval of several different cargo proteins from the endosome to the " trans "-Golgi network.
47.
Phosphatidylinositol 3-phosphate, a common endosomal lipid, binds to this FYVE domain resulting in the recruitment of ESCRT-0 to the endosome.
48.
These changes, as well as the toxic nature of the pentons, destroy the endosome, resulting in the movement of the virion into the cytoplasm.
49.
In general, exosomes are formed by segregating the cargo ( e . g ., lipids, proteins, and nucleic acids ) within the endosome.
50.
The late endosome itself can eventually grow into a mature lysosome, as evidenced by the transport of endosomal membrane components from the lysosomes back to the endosomes.
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