EAD is an abnormal depolarization and increase in action potential frequency that occurs in cardiac myocytes before normal repolarization is complete.
42.
This shortens the repolarization period, thus speeding the rate of depolarization and contraction, which results in an increased heart rate.
43.
Class III agents ( prolong repolarization by blocking outward K + current ) : amiodarone and sotalol are effective class III agents.
44.
This gene encodes a protein for a voltage-gated potassium channel required for the repolarization phase of the cardiac action potential.
45.
The QT interval represents ventricular depolarization and repolarization, and XEN-D0101 did not significantly increase the timing of this interval.
46.
The QT interval as recorded on a standard electrocardiogram ( EKG ) represents ventricular depolarization and ventricular repolarization and is rate-dependent.
47.
In addition, the reduced extracellular potassium ( paradoxically ) inhibits the activity of the I Kr potassium current and delays ventricular repolarization.
48.
There also have been some studies that suggest that cicutoxin increases the duration of the neuronal repolarization in a dose-dependent manner.
49.
The two most common clinical conditions where ECG findings may mimic pericarditis are acute myocardial infarction ( AMI ) and generalized early repolarization.
50.
In mouse cardiomyocytes, K v 2.1 channel is the molecular substrate of major repolarization current I K-slow2 . arrhythmia.
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