hypervitaminosis sentence in Hindi
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- The cause of Mertz's death has never been firmly established; the commonly purported theory is hypervitaminosis A ( an excessive intake of vitamin A ) from consuming the livers of the Huskies.
- Elevated amounts of retinyl ester ( i . e ., > 10 % of total circulating vitamin A ) in the fasting state have been used as markers for chronic hypervitaminosis A in humans.
- Elevated amounts of retinyl ester ( i . e ., > 10 % of total circulating vitamin A ) in the fasting state have been used as markers for chronic hypervitaminosis A in humans and monkeys.
- De Veer kept a diary of the voyages and in 1597 was the first person to observe and record the Novaya Zemlya effect, and the first westerner to observe hypervitaminosis A caused by consuming polar bear liver.
- "' Hypervitaminosis A "'refers to the toxic effects of ingesting too much preformed vitamin A . Symptoms arise as a result of altered bone metabolism and altered metabolism of other fat-soluble vitamins.
- High intake of provitamin carotenoids ( such as beta carotene ) from vegetables and fruits does not cause hypervitaminosis A, as conversion from carotenoids to the active form of vitamin A is regulated by the body to maintain an optimum level of the vitamin.
- Hypervitaminosis A can be prevented by not ingesting more than the US Institute of Medicine Daily Tolerable Upper Level of intake for Vitamin A . This level is for synthetic and natural retinol ester forms of vitamin A . Carotene forms from dietary sources are not toxic.
- A 1969 study by depression, dysentery and persistent skin infections indicated the men had suffered hypervitaminosis A, an excessive intake of vitamin A . This is found in unusually high quantities in the livers of Greenland Huskies, of which both Mertz and Mawson consumed large amounts.
- The liver of one dog contains enough vitamin A to produce the condition called Hypervitaminosis A . Mertz became incapacitated and incoherent; in an attempt to nurse him back to health, Mawson fed him most of the dog livers, which he considered more nourishing than the tough muscle tissue.
- Too much vitamin A in retinoid form can be harmful or fatal, resulting in what is known as hypervitaminosis A . The body converts the dimerized form, carotene, into vitamin A as it is needed, therefore high levels of carotene are not toxic compared to the ester ( animal ) forms.
- This change in bone turnover is likely to be the reason for numerous effects seen in hypervitaminosis A, such as hypercalcemia and numerous bone changes such as bone loss that potentially leads to osteoporosis, spontaneous bone fractures, altered skeletal development in children, skeletal pain, radiographic changes, and bone lesions.
- The causes of Mertz's death and Mawson's related illness remain uncertain; a 1969 study suggested hypervitaminosis A, presumably caused by the men eating the livers of their Sir Edmund Hillary described Mawson's month-long journey as " probably the greatest story of lone survival in Polar exploration ".
- With six dogs between them ( with a liver on average weighing 1 kg ), it is thought that the pair ingested enough liver to bring on a condition known as Hypervitaminosis A . However, Mertz may have suffered more because he found the tough muscle tissue difficult to eat and therefore ate more of the liver than Mawson . While both men suffered, Mertz suffered more severely.
- You don't have to predicate a theory of this sort to explain the soles coming off your feet . " A 2005 article in " The Medical Journal of Australia " by Denise Carrington-Smith suggested it may have been " the psychological stresses related to the death of a close friend and the deaths of the dogs he had cared for ", and a switch from a predominately vegetarian diet that killed Mertz, not hypervitaminosis A.
- Apart from this paper, in this period, Hugh had published on a variety of topics, including nutritional deficiency in general and in more specific aspects, such as the relation of deficiencies of vitamin A and essential fatty acids to follicular hyperkeratosis in rats, lesions of mucocutaneous zones in the rat in pyridoxine deficiency, ascorbic acid in hypervitaminosis A in guinea pigs, polycythemia in the rat in pyridoxine deficiency, skin permeability in deficiency of essential fatty acids, vitamin deficiencies in alcoholism, vitamins and the nervous system, and nutritional neuropathy in chronic thiamine deficiency in the rat.
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