Atherogenesis is the developmental process of atheromatous plaques.
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It is where small atheromatous plaques build up in the walls of medium and large arteries.
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Statins are the most frequently used lipid / cholesterol modifiers, which probably also stabilize existing atheromatous plaque.
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In contrast, having small amounts of large HDL particles is independently associated with atheromatous disease progression within the arteries.
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The muscular portion of artery walls usually remain strong, even after they have remodeled to compensate for the atheromatous plaques.
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In the context of heart or artery matters, atheromata are commonly referred to as atheromatous "'plaques " '.
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M2 macrophages can remove cholesterol from blood vessels, but when the cholesterol is oxidized, the M2 macrophages become atheromatous plaque of atherosclerosis.
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In contrast, large numbers of small dense LDL ( sdLDL ) particles are strongly associated with the presence of atheromatous disease within the arteries.
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Kovanen PT, Kaartinen M, Paavonen T . Infiltrates of activated mast cells at the site of coronary atheromatous erosion or rupture in myocardial infarction.
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Large numbers of HDL particles correlates with better health outcomes ., whereas low numbers of HDL particles is associated with atheromatous disease progression in the arteries.
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