Erythropoietic porphyria is a genetic disease resulting in the deposition of porphyrins throughout the body.
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Defects in ferrochelatase create a buildup of protoporphyrin IX, causing erythropoietic protoporphyria ( EPP ).
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A deficiency is associated with Gunther's disease, also known as congenital erythropoietic porphyria ( CEP ).
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The pain, burning, swelling, and itching that occur in erythropoietic porphyrias generally require avoidance of bright sunlight.
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The rarest is congenital erythropoietic porphyria ( C . E . P ), otherwise known as Gunther's disease.
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Erythropoietin is the primary erythropoietic factor that cooperates with various other growth factors ( e . g ., multipotent progenitors.
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It should also be noted that quite a bit of research emphesis is on non erythropoietic but, neuroprotective Peptides of Erythropoietin.
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This suggests a possible extra-ribosomal function for this gene in erythropoietic differentiation and proliferation, in addition to its ribosomal function.
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Peptide of Epo with amino acids 92-111 is neuroprotective while its erythropoietic potency is 10 fold less than the wild type.
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The U . S . Food and Drug Administration have recognized afamelanotide as an orphan drug for the treatment of EPP and congenital erythropoietic porphyria.
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