myocyte sentence in Hindi
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- These changes impair mitochondrial energy production and drive cardiac myocyte apoptosis.
- GATA4 and GATA6 also play a role in myocyte differentiation.
- TEAD proteins and MEF2 ( myocyte enhancer factor 2 ) interact physically.
- These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation.
- This means that the refractoriness of the ventricular myocyte increases at lower heart rates.
- The electrical depolarizations that trigger cardiac myocytes to contract arise spontaneously within the myocyte itself.
- One TC establishing contacts with a myocyte by a Tp of about 65 mm long.
- The cardiac conduction system ( and AV node part of it ) coordinates myocyte mechanical activity.
- Excitation of a myocyte causes depolarization at its synapses, the neuromuscular junctions, which triggers action potential.
- ITM2A is also involved in activation of T-cells in the immune system and in myocyte differentiation.
- Furthermore, FAK is also involved in the maintenance of sarcomeric organization, cell survival, and myocyte myocyte interactions.
- A second family of DNA binding regulatory proteins is the myocyte-specific enhancer factor-2 ( MEF2 ) family.
- Not only was there an absence of HCM, but fibrosis and myocyte disorganization was greatly reduced in the knockdown mice.
- As the heart fills with more blood than usual, there is an increase in the load experienced by each myocyte.
- The "'diad "'is a structure in the cardiac myocyte located at the sarcomere Z-line.
- OPN expression increases under a variety of conditions of the heart, and is associated with increased myocyte apoptosis and myocardial dysfunction.
- It also interacts with myocyte enhancer factor-2 ( MEF2 ) proteins, resulting in repression of MEF2-dependent genes.
- Without the dysfunctional myosin protein the heart functioned more efficiently and this prevents the development of myocyte hypertrophy as a compensatory mechanism.
- Deletion of Kcne4 in mice impaired ventricular myocyte Ito, a current generated at least in part by K v 4.2.
- Like skeletal myocytes ( and axons for that matter ), in the resting state a given cardiac myocyte has a negative membrane potential.
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