Males with embryonal carcinoma tend to have a normal range serum AFP . The finding of elevated AFP is more suggestive of a mixed germ cell tumour, with the AFP being released by a yolk sac tumour component.
32.
Because undifferentiated pluripotent stem cells have elevated levels of alkaline phosphatase on their cell membrane, therefore alkaline phosphatase staining is used to detect these cells and to test pluripotency ( i . e ., embryonic stem cells or embryonal carcinoma cells ).
33.
The Cas family comprises other three members : NEDD9 ( Neural precursor cell expressed, developmentally down-regulated 9, also called Human enhancer of filamentation 1, EFS ( Embryonal Fyn-associated substrate ), and CASS4 ( Cas scaffolding protein family member 4 ).
34.
The first study of perlecan expression during embryonal development found that the protein was first expressed during development of the cardiovascular system, and later correlates with maturation of the majority of tissues in the body, i . e . separation of epithelial layers from endothelia and stroma by basement membranes.
35.
In embryonal development, both lung and swim bladder originate as an outpocketing from the gut; in the case of swim bladders, this connection to the gut continues to exist as the pneumatic duct in the more " primitive " ray-finned fish, and is lost in some of the more derived teleost orders.
36.
EFS ( E mbryonal F yn-associated S ubstrate ), also known as SIN ( S rc IN teracting or S ignal Integrating protein ) was originally identified using cDNA library screening of mouse embryonal libraries for proteins containing SRC SH3 domain, in two independent studies by Ishino et al . in 1995 and Alexandropoulos et al . in 1996.
37.
However, it is unclear whether the mutation of the genes is directly related to the development of RMS . One study showed a that 35 % of embryonal RMS tumors contained activating mutations in either N-or K-" ras, " and it is worth noting that " ras " activation has been shown to block myogenic differentiation, which could help explain its potential role in rhabdomyosarcogenesis.
38.
It red a 2.2-fold increase in leukemias and a 1.6-fold increase in solid ( mainly embryonal ) cancers among children living within 5 km of all German nuclear power stations . " In 2011 a new study of the KiKK data was incorporated into an assessment by the Committee on Medical Aspects of Radiation in the Environment ( COMARE ) of the incidence of childhood leukemia around British nuclear power plants.
39.
:: : ADD has at least partly genetic causes per current consensus, but it's hard to tell which is more important; I lean towards genetic factors being slightly more important at least ( of course there are factors which are outside the strict genetic environmental dichotomy, such as in embryonal development, which can be described as innate but not necessarily genetically caused, though possibly influenced by genetic factors, despite apparent environmental causes; consider that an alcohol-addicted mother can cause harm to her child's development, possibly resulting in mental issues, while her own addiction may have biological factors contributing to its rise, for example by developping a mental instability that makes her react considerably more sensitively to setbacks this seriously complicates things : if the child develops ADD, this may seem to be caused by the mother's alcohol abuse, but in fact the tendency towards the development of ADD may simply, or additionally, be inherited from the mother, making cause and effect hard to disentangle ).
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