The " P . falciparum " aldolase is a 41 kDa protein and has 61-68 % sequence similarity to known eukaryotic aldolases.
42.
The aldol cleavage of fructose 1, 6-bisphosphate by aldolase b demonstrates the different reaction products, dihydroxyacetone phosphate and glyceraldehyde 3-phosphate.
43.
The first recorded case of Aldolase A deficiency was described in 1973 ( Beutler et al . ) of a Jewish Canadian boy of Romanian descent.
44.
In human liver, purified fructokinase, when coupled with aldolase, has been discovered to contribute to an alternative mechanism to produce oxalate from xylitol.
45.
In coupled sequence, fructokinase and aldolase produce glycolaldehyde, a precursor to oxalate, from D-xylulose via D-xylulose 1-phosphate.
46.
Due to the lack of functional aldolase B, organisms with HFI cannot properly process F1P, which leads to an accumulation of F1P in bodily tissues.
47.
The low incidence of this syndrome is often related to aldolase A's essential glycolytic role along with its exclusive expression in blood and skeletal muscle.
48.
It is isomerized to 5-fluoro-5-deoxy-ribulose-1-phosphate which is then cleaved by an aldolase to release fluoroacetaldehyde.
49.
Other markers of muscle damage, such as aldolase, troponin, fatty acid-binding protein ( FABP ), are mainly used in chronic muscle diseases.
50.
Biochemical investigations include serum creatine kinase ( typically increased 10 fold ) with lesser elevations of the serum aldolase, aspartate transaminase, alanine transaminase and lactic dehydrogenase.
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